TMEM161A 抗体 (AA 21-120) (AbBy Fluor® 350)
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- 抗原 See all TMEM161A products
- TMEM161A (Transmembrane Protein 161A (TMEM161A))
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抗原表位
- AA 21-120
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适用
- 人
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宿主
- 兔
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克隆类型
- 多克隆
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标记
- This TMEM161A antibody is conjugated to AbBy Fluor® 350
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应用范围
- Western Blotting (WB), Immunofluorescence (Cultured Cells) (IF (cc)), Immunofluorescence (Paraffin-embedded Sections) (IF (p))
- 交叉反应
- 人
- 预测反应
- Mouse,Rat,Dog,Cow,Sheep,Pig,Horse,Rabbit
- 纯化方法
- Purified by Protein A.
- 免疫原
- KLH conjugated synthetic peptide derived from human TMEM161A
- 亚型
- IgG
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- 应用备注
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IF(IHC-P) 1:50-200
IF(IHC-F) 1:50-200
IF(ICC) 1:50-200 - 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 μg/μL
- 缓冲液
- Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.
- 储存液
- ProClin
- 注意事项
- This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C. Aliquot into multiple vials to avoid repeated freeze-thaw cycles.
- 有效期
- 12 months
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- 抗原
- TMEM161A (Transmembrane Protein 161A (TMEM161A))
- 别名
- TMEM161A (TMEM161A 产品)
- 别名
- AROS-29 antibody, AI428876 antibody, BB161850 antibody, BC021367 antibody, RGD1307703 antibody, transmembrane protein 161A antibody, transmembrane protein 161A L homeolog antibody, TMEM161A antibody, Tmem161a antibody, tmem161a.L antibody
- 背景
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Synonyms: AROS-29, T161A_HUMAN, tmem161a, Transmembrane protein 161A.
Background: AROS-29 is suggested to have a functional role in protection against oxidative stress. The gene encoding AROS-29 is located on human chromosome 19, which consists of over 63 million bases, houses approximately 1,400 genes and is recognized for having the greatest gene density of the human chromosomes. It is the genetic home for a number of immunoglobulin superfamily members, including the killer cell and leukocyte Ig-like receptors, a number of ICAMs, the CEACAM and PSG family and Fc receptors (FcRs). Key genes for eye color and hair color also map to chromosome 19.
- 基因ID
- 54929
- 途径
- Positive Regulation of Response to DNA Damage Stimulus
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