Angiomotin 抗体 (AA 545-620)
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- 抗原 See all Angiomotin (AMOT) 抗体
- Angiomotin (AMOT)
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抗原表位
- AA 545-620
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适用
- 人
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宿主
- 兔
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克隆类型
- 多克隆
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标记
- This Angiomotin antibody is un-conjugated
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应用范围
- Western Blotting (WB), ELISA, Immunohistochemistry (Paraffin-embedded Sections) (IHC (p)), Immunofluorescence (Paraffin-embedded Sections) (IF (p)), Immunohistochemistry (Frozen Sections) (IHC (fro)), Immunofluorescence (Cultured Cells) (IF (cc))
- 交叉反应
- 人
- 预测反应
- Mouse,Rat,Dog,Cow,Pig,Horse
- 纯化方法
- Purified by Protein A.
- 免疫原
- KLH conjugated synthetic peptide derived from human Angiomotin
- 亚型
- IgG
- Top Product
- Discover our top product AMOT Primary Antibody
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- 应用备注
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WB 1:300-5000
ELISA 1:500-1000
IHC-P 1:200-400
IHC-F 1:100-500
IF(IHC-P) 1:50-200
IF(IHC-F) 1:50-200
IF(ICC) 1:50-200 - 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 μg/μL
- 缓冲液
- 0.01M TBS( pH 7.4) with 1 % BSA, 0.02 % Proclin300 and 50 % Glycerol.
- 储存液
- ProClin
- 注意事项
- This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
- 储存条件
- 4 °C,-20 °C
- 储存方法
- Shipped at 4°C. Store at -20°C for one year. Avoid repeated freeze/thaw cycles.
- 有效期
- 12 months
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Loss of tricellular tight junction protein LSR promotes cell invasion and migration via upregulation of TEAD1/AREG in human endometrial cancer." in: Scientific reports, Vol. 7, pp. 37049, (2017) (PubMed).
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Loss of tricellular tight junction protein LSR promotes cell invasion and migration via upregulation of TEAD1/AREG in human endometrial cancer." in: Scientific reports, Vol. 7, pp. 37049, (2017) (PubMed).
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- 抗原
- Angiomotin (AMOT)
- 别名
- Angiomotin (AMOT 产品)
- 别名
- C81439 antibody, CAG-2 antibody, D0Kist1 antibody, E230009N18Rik antibody, Sii6 antibody, mKIAA1071 antibody, RGD1564027 antibody, si:dkey-13f9.6 antibody, wu:fj21a07 antibody, angiomotin antibody, microRNA 4329 antibody, AMOT antibody, Amot antibody, MIR4329 antibody, amot antibody
- 背景
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Synonyms: Angiomotin, AMOT, KIAA171
Background: Plays a central role in tight junction maintenance via the complex formed with ARHGAP17, which acts by regulating the uptake of polarity proteins at tight junctions. Appears to regulate endothelial cell migration and tube formation. May also play a role in the assembly of endothelial cell-cell junctions.
- 基因ID
- 154796
- UniProt
- Q4VCS5
- 途径
- Cell-Cell Junction Organization, Regulation of Cell Size
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