MOCOS 抗体
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- 抗原 See all MOCOS 抗体
- MOCOS (Molybdenum Cofactor Sulfurase (MOCOS))
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适用
- 人
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宿主
- 兔
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克隆类型
- 多克隆
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标记
- This MOCOS antibody is un-conjugated
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应用范围
- Western Blotting (WB)
- 产品特性
- Polyclonal Antibody
- 纯化方法
- Affinity purification
- 免疫原
- Recombinant fusion protein of human MOCOS (NP_060417.2).
- 亚型
- IgG
- Top Product
- Discover our top product MOCOS Primary Antibody
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- 应用备注
- WB 1:500-1:2000
- 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 mg/mL
- 缓冲液
- PBS with 0.02 % sodium azide, 50 % glycerol, pH 7.3
- 储存液
- Sodium azide
- 注意事项
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C. Avoid freeze / thaw cycles.
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- 抗原
- MOCOS (Molybdenum Cofactor Sulfurase (MOCOS))
- 别名
- MOCOS (MOCOS 产品)
- 别名
- HMCS antibody, MCS antibody, MOS antibody, 1110018O12Rik antibody, DDBDRAFT_0217046 antibody, DDBDRAFT_0252757 antibody, DDB_0217046 antibody, DDB_0252757 antibody, Og antibody, im:7142948 antibody, mocos antibody, molybdenum cofactor sulfurase antibody, MOCOS antibody, Mocos antibody, AOR_1_1090144 antibody, mocos antibody, MCYG_05594 antibody, VDBG_04857 antibody, MGYG_02684 antibody, TERG_06775 antibody, mal antibody
- 背景
- This gene encodes an enzyme that sulfurates the molybdenum cofactor which is required for activation of the xanthine dehydrogenase (XDH) and aldehyde oxidase (AO) enzymes. XDH catalyzes the conversion of hypoxanthine to uric acid via xanthine, as well as the conversion of allopurinol to oxypurinol, and pyrazinamide to 5-hydroxy pyrazinamide. Mutations in this gene cause the metabolic disorder classical xanthinuria type II which is characterized by the loss of XDH/XO and AO enzyme activity, decreased levels of uric acid in the urine, increased levels of xanthine and hypoxanthine in the serum and urine, formation of xanthine stones in the urinary tract, and myositis due to tissue deposition of xanthine.
- 分子量
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Observed_MW: 98 kDa
Calculated_MW: 98 kDa
- 基因ID
- 55034
- UniProt
- Q96EN8
- 途径
- Response to Water Deprivation, ER-Nucleus Signaling
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