-
- 抗原 See all TGFBI 抗体
- TGFBI (Transforming Growth Factor, beta-Induced, 68kDa (TGFBI))
-
适用
- 人, 小鼠, 大鼠
-
宿主
- 兔
-
克隆类型
- 多克隆
-
标记
- This TGFBI antibody is un-conjugated
-
应用范围
- Western Blotting (WB), Immunohistochemistry (IHC), ELISA
- 产品特性
- Polyclonal Antibody
- 纯化方法
- Antigen affinity purification
- 免疫原
- Fusion protein of human TGFBI
- 亚型
- IgG
- Top Product
- Discover our top product TGFBI Primary Antibody
-
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 481-683) antibody
TGFBI 适用: 人 WB, IHC, IF 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 423-632) antibodyTGFBI 适用: 人 WB, IHC, IP, ICC 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 1-683) antibodyTGFBI 适用: 人 WB 宿主: 小鼠 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 1-683) antibodyTGFBI 适用: 人 WB, IP 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (Internal Region) antibodyVerified TGFBI 适用: 人, 大鼠 WB, IHC, ELISA, IF 宿主: 山羊 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 481-580) antibodyTGFBI 适用: 人 WB, ELISA 宿主: 小鼠 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 423-632) antibodyTGFBI 适用: 小鼠 WB, IHC, IP, ICC 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 30-250) antibodyTGFBI 适用: 人 IHC, ELISA 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (AA 601-650) antibodyTGFBI 适用: 人, 小鼠, 犬, Cow, 马, 兔, 豚鼠 WB, IHC, IHC (p) 宿主: 兔 Polyclonal unconjugated
anti-Transforming Growth Factor, beta-Induced, 68kDa (TGFBI) (N-Term) antibodyTGFBI 适用: 人, 小鼠, 大鼠 WB, IHC, ELISA, ICC, IF 宿主: 兔 Polyclonal unconjugated
-
- 应用备注
- WB 1:500-1:2000, IHC 1:50-1:200, ELISA 1:5000-1:10000
- 限制
- 仅限研究用
-
- 状态
- Liquid
- 浓度
- 1.02 mg/mL
- 缓冲液
- PBS with 0.05 % Sodium azide and 40 % Glycerol, pH 7.4
- 储存液
- Sodium azide
- 注意事项
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C. Avoid freeze / thaw cycles.
-
- 抗原
- TGFBI (Transforming Growth Factor, beta-Induced, 68kDa (TGFBI))
- 别名
- TGFBI (TGFBI 产品)
- 别名
- TGFBI antibody, BIGH3 antibody, CDB1 antibody, CDG2 antibody, CDGG1 antibody, CSD antibody, CSD1 antibody, CSD2 antibody, CSD3 antibody, EBMD antibody, LCD1 antibody, 68kDa antibody, AI181842 antibody, AI747162 antibody, Beta-ig antibody, big-h3 antibody, Big-h3 antibody, MP70 antibody, MP78 antibody, hm:zeh0933 antibody, wu:fa13f05 antibody, zgc:110495 antibody, transforming growth factor beta induced antibody, transforming growth factor, beta induced antibody, transforming growth factor, beta-induced antibody, TGFBI antibody, Tgfbi antibody, tgfbi antibody
- 背景
- TGFBI,also named as BIGH3,Kerato-epithelin and RGD-CAP,binds to type I,II,and IV collagens. TGFBI is an adhesion protein which may play an important role in cell-collagen interactions. In cartilage,it may be involved in endochondral bone formation. TGFBI is an extracellular matrix adaptor protein,it has been reported to be differentially expressed in transformed tissues. TGFBI is a predictive factor of the response to chemotherapy,and suggest the use of TGFBI-derived peptides as possible therapeutic adjuvants for the enhancement of responses to chemotherapy. Defects in TGFBI are the cause of epithelial basement membrane corneal dystrophy (EBMD). Defects in TGFBI are the cause of corneal dystrophy Groenouw type 1 (CDGG1). Defects in TGFBI are the cause of corneal dystrophy lattice type 1 (CDL1). Defects in TGFBI are a cause of corneal dystrophy Thiel-Behnke type (CDTB). Defects in TGFBI are the cause of Reis-Buecklers corneal dystrophy (CDRB). Defects in TGFBI are the cause of lattice corneal dystrophy type 3A (CDL3A). Defects in TGFBI are the cause of Avellino corneal dystrophy (ACD).
- 分子量
-
Observed_MW: Refer to figures
Calculated_MW: 75 kDa
- UniProt
- Q15582
-
You are here:
