SDCBP 抗体 (AA 1-100)
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- 抗原 See all SDCBP 抗体
- SDCBP (Syndecan Binding Protein (Syntenin) (SDCBP))
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抗原表位
- AA 1-100
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适用
- 人
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宿主
- 小鼠
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克隆类型
- 单克隆
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标记
- This SDCBP antibody is un-conjugated
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应用范围
- Western Blotting (WB), ELISA, Immunofluorescence (IF), Immunoprecipitation (IP), Immunohistochemistry (Paraffin-embedded Sections) (IHC (p))
- 原理
- Mouse monoclonal antibody raised against a partial recombinant SDCBP.
- 序列
- MSLYPSLEDL KVDKVIQAQT AFSANPANPA ILSEASAPIP HDGNLYPRLY PELSQYMGLS LNEEEIRANV AVVSGAPLQG QLVARPSSIN YMVAPVTGND
- 交叉反应
- 人
- 产品特性
- Antibody Reactive Against Recombinant Protein.
- 免疫原
- SDCBP (NP_005616, 1 a.a. ~ 100 a.a) partial recombinant protein with GST tag. MW of the GST tag alone is 26 KDa.
- 克隆位点
- 2C12
- 亚型
- IgG1
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- 应用备注
- Optimal working dilution should be determined by the investigator.
- 限制
- 仅限研究用
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- 缓冲液
- In 1x PBS, pH 7.4
- 注意事项
- Aliquot to avoid repeated freezing and thawing.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
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Syntenin increases the invasiveness of small cell lung cancer cells by activating p38, AKT, focal adhesion kinase and SP1." in: Experimental & molecular medicine, Vol. 46, pp. e90, (2014) (PubMed).
: "MDA-9/syntenin and IGFBP-2 promote angiogenesis in human melanoma." in: Cancer research, Vol. 73, Issue 2, pp. 844-54, (2013) (PubMed).
: "Novel role of MDA-9/syntenin in regulating urothelial cell proliferation by modulating EGFR signaling." in: Clinical cancer research : an official journal of the American Association for Cancer Research, Vol. 19, Issue 17, pp. 4621-33, (2013) (PubMed).
: "Raf kinase inhibitor RKIP inhibits MDA-9/syntenin-mediated metastasis in melanoma." in: Cancer research, Vol. 72, Issue 23, pp. 6217-26, (2012) (PubMed).
: "Systematic proteomic analysis of human hepotacellular carcinoma cells reveals molecular pathways and networks involved in metastasis." in: Molecular bioSystems, Vol. 7, Issue 6, pp. 1908-16, (2011) (PubMed).
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Syntenin increases the invasiveness of small cell lung cancer cells by activating p38, AKT, focal adhesion kinase and SP1." in: Experimental & molecular medicine, Vol. 46, pp. e90, (2014) (PubMed).
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