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CXCL9 抗体 (Alexa Fluor 647)

CXCL9 适用: 小鼠 FACS 宿主: Armenian Hamster Monoclonal MIG-2F5-5 Alexa Fluor 647
产品编号 ABIN2657882
发货至: 中国
  • 抗原 See all CXCL9 抗体
    CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))
    适用
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    小鼠
    宿主
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    Armenian Hamster
    克隆类型
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    单克隆
    标记
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    This CXCL9 antibody is conjugated to Alexa Fluor 647
    应用范围
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    Flow Cytometry (FACS)
    纯化方法
    The antibody was purified by affinity chromatography, and conjugated with Alexa Fluor® 647 under optimal conditions.
    克隆位点
    MIG-2F5-5
    亚型
    IgG, IgG kappa
    Top Product
    Discover our top product CXCL9 Primary Antibody
  • 应用备注
    Optimal working dilution should be determined by the investigator.
    限制
    仅限研究用
  • 浓度
    0.5 mg/mL
    缓冲液
    Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide.
    储存液
    Sodium azide
    注意事项
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    注意事项
    Protect from prolonged exposure to light. Do not freeze.
    储存条件
    4 °C
    储存方法
    The antibody solution should be stored undiluted between 2°C and 8°C
  • 抗原
    CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))
    别名
    CXCL9 (CXCL9 产品)
    别名
    CMK antibody, Humig antibody, MIG antibody, SCYB9 antibody, crg-10 antibody, BB139920 antibody, Mig antibody, MuMIG antibody, Scyb9 antibody, C-X-C motif chemokine ligand 9 antibody, chemokine (C-X-C motif) ligand 9 antibody, CXCL9 antibody, Cxcl9 antibody
    背景
    MIG, also known as mig-1, CXCL9, is a member of the alpha subfamily of inflammatory chemokine. It is inducible in macrophages, hepatocytes, and endothelial cells by IFN-γ, but not by TNF-α or bacterial lipopolysacchrides (LPS). Mig functions as a chemotactic factor for resting memory and activated T cells, both CD4+ and CD8+, and natural killer cells. Furthermore, it was reported that Mig induced both calcium signals and chemotaxis in activated B cells and that B cell activation induced expression of mouse CXCR3. MIG and CXCR3 may be important not only to recruit T cells to peripheral inflammatory sites, but also in some cases to maximize interactions among activated T cells, B cells, and dendritic cells within lymphoid organs to provide optimal humoral responses to pathogens.
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