CXCL9 抗体 (Alexa Fluor 647)
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- 抗原 See all CXCL9 抗体
- CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))
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适用
- 小鼠
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宿主
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Armenian Hamster
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克隆类型
- 单克隆
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标记
- This CXCL9 antibody is conjugated to Alexa Fluor 647
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应用范围
- Flow Cytometry (FACS)
- 纯化方法
- The antibody was purified by affinity chromatography, and conjugated with Alexa Fluor® 647 under optimal conditions.
- 克隆位点
- MIG-2F5-5
- 亚型
- IgG, IgG kappa
- Top Product
- Discover our top product CXCL9 Primary Antibody
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- 应用备注
- Optimal working dilution should be determined by the investigator.
- 限制
- 仅限研究用
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- 浓度
- 0.5 mg/mL
- 缓冲液
- Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide.
- 储存液
- Sodium azide
- 注意事项
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- 注意事项
- Protect from prolonged exposure to light. Do not freeze.
- 储存条件
- 4 °C
- 储存方法
- The antibody solution should be stored undiluted between 2°C and 8°C
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- 抗原
- CXCL9 (gamma-Interferon-Induced Monokine (CXCL9))
- 别名
- CXCL9 (CXCL9 产品)
- 别名
- CMK antibody, Humig antibody, MIG antibody, SCYB9 antibody, crg-10 antibody, BB139920 antibody, Mig antibody, MuMIG antibody, Scyb9 antibody, C-X-C motif chemokine ligand 9 antibody, chemokine (C-X-C motif) ligand 9 antibody, CXCL9 antibody, Cxcl9 antibody
- 背景
- MIG, also known as mig-1, CXCL9, is a member of the alpha subfamily of inflammatory chemokine. It is inducible in macrophages, hepatocytes, and endothelial cells by IFN-γ, but not by TNF-α or bacterial lipopolysacchrides (LPS). Mig functions as a chemotactic factor for resting memory and activated T cells, both CD4+ and CD8+, and natural killer cells. Furthermore, it was reported that Mig induced both calcium signals and chemotaxis in activated B cells and that B cell activation induced expression of mouse CXCR3. MIG and CXCR3 may be important not only to recruit T cells to peripheral inflammatory sites, but also in some cases to maximize interactions among activated T cells, B cells, and dendritic cells within lymphoid organs to provide optimal humoral responses to pathogens.
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