Adracalin 抗体 (AA 51-150) (AbBy Fluor® 647)
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- 抗原 See all Adracalin (AAAS) 抗体
- Adracalin (AAAS) (Achalasia, Adrenocortical Insufficiency, Alacrimia (AAAS))
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抗原表位
- AA 51-150
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适用
- 人
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宿主
- 兔
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克隆类型
- 多克隆
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标记
- This Adracalin antibody is conjugated to AbBy Fluor® 647
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应用范围
- Western Blotting (WB), Immunofluorescence (Cultured Cells) (IF (cc)), Immunofluorescence (Paraffin-embedded Sections) (IF (p))
- 预测反应
- Human,Mouse,Rat,Cow
- 纯化方法
- Purified by Protein A.
- 免疫原
- KLH conjugated synthetic peptide derived from human Adracalin
- 亚型
- IgG
- Top Product
- Discover our top product AAAS Primary Antibody
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- 应用备注
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IF(IHC-P) 1:50-200
IF(IHC-F) 1:50-200
IF(ICC) 1:50-200 - 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 μg/μL
- 缓冲液
- Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.
- 储存液
- ProClin
- 注意事项
- This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C. Aliquot into multiple vials to avoid repeated freeze-thaw cycles.
- 有效期
- 12 months
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- 抗原
- Adracalin (AAAS) (Achalasia, Adrenocortical Insufficiency, Alacrimia (AAAS))
- 别名
- Adracalin (AAAS 产品)
- 别名
- zgc:85873 antibody, aladin antibody, Aladin antibody, AAA antibody, AAASb antibody, ADRACALA antibody, ADRACALIN antibody, ALADIN antibody, D030041N15Rik antibody, GL003 antibody, aladin WD repeat nucleoporin antibody, achalasia, adrenocortical insufficiency, alacrimia antibody, aladin WD repeat nucleoporin L homeolog antibody, Aaas antibody, aaas antibody, aaas.L antibody, AAAS antibody
- 背景
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Synonyms: AAA, AAAS, AAASb, Achalasia adrenocortical insufficiency alacrimia Allgrove triple A, Achalasia adrenocortical insufficiency alacrimia, ADRACALA, Aladin, Allgrove triple A, DKFZp586G1624, GL003, AAAS_HUMAN.
Background: Aladin (Adracalin) belongs to a family of WD repeat-containing proteins. These proteins have a wide variety of functions, including signal transduction regulation, RNA processing and transcription. Aladin plays a role in peripheral and central nervous system development. It is widely expressed, with the highest expression seen in pituitary gland, corpus callosum, cerebellum, adrenal gland and gastrointestinal structures. Defects in Aladin cause the autosomal recessive disorder achalasia-addisonianism-alacrima (triple A) syndrome. Triple A syndrome is characterized by achalasia, alacrima and adrenocortico-tropin-resistant adrenal insufficiency. Robust expression in neural systems associated with cognitive, motor and sensory functions is consistent with the myriad of symptoms experienced by patients with triple A syndrome.
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