AFF2 抗体 (AA 1-80) (AbBy Fluor® 350)
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- 抗原 See all AFF2 抗体
- AFF2 (AF4/FMR2 Family, Member 2 (AFF2))
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抗原表位
- AA 1-80
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适用
- 人
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宿主
- 兔
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克隆类型
- 多克隆
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标记
- This AFF2 antibody is conjugated to AbBy Fluor® 350
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应用范围
- Western Blotting (WB), Immunofluorescence (Cultured Cells) (IF (cc)), Immunofluorescence (Paraffin-embedded Sections) (IF (p))
- 预测反应
- Human,Mouse,Rat,Dog,Cow,Pig,Horse
- 纯化方法
- Purified by Protein A.
- 免疫原
- KLH conjugated synthetic peptide derived from human AFF2
- 亚型
- IgG
- Top Product
- Discover our top product AFF2 Primary Antibody
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- 应用备注
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IF(IHC-P) 1:50-200
IF(IHC-F) 1:50-200
IF(ICC) 1:50-200 - 限制
- 仅限研究用
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- 状态
- Liquid
- 浓度
- 1 μg/μL
- 缓冲液
- Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.
- 储存液
- ProClin
- 注意事项
- This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE, which should be handled by trained staff only.
- 储存条件
- -20 °C
- 储存方法
- Store at -20°C. Aliquot into multiple vials to avoid repeated freeze-thaw cycles.
- 有效期
- 12 months
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- 抗原
- AFF2 (AF4/FMR2 Family, Member 2 (AFF2))
- 别名
- FMR2/AFF2 (AFF2 产品)
- 别名
- FMR2 antibody, FMR2P antibody, FRAXE antibody, MRX2 antibody, OX19 antibody, Fmr2 antibody, Ox19 antibody, Oxh antibody, AF4/FMR2 family member 2 antibody, AF4/FMR2 family, member 2 antibody, AFF2 antibody, Aff2 antibody
- 背景
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Synonyms: AF4/FMR2 family member 2, AF4/FMR2 family, member 2, AFF2, AFF2_HUMAN, FMR2, FMR2P, Fragile X E mental retardation syndrome protein, fragile X mental retardation 2, Fragile X mental retardation 2 protein, fragile X mental retardation gene associated with FRAXE, FRAXE, mild or borderline mental retardation, MRX2, OX19, Protein FMR-2, Protein Ox19.
Background: FMR2 is a 1311 amino acid nuclear protein belonging to the AF4 family. Expressed in the brain, placenta and lung, FMR2 exists as two isoforms produced by alternative splicing. Defects in the gene that encodes FMR2 have been found to be a cause of FRAXE, an X-linked form of mental retardation. Individuals expressing the FRAXE site also have more than two-hundred copies of a GCC repeat adjacent to CpG island, compared to six to thirty-five copies of the GCC repeat in a normal individual. It is believed that loss of FMR2 expression causes this GCC expansion of the FRAXE site.
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